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Re: kohlenhydrate/reiche /arme ernährung [Beitrag #16828 ist eine Antwort auf Beitrag #16802] :: Di., 28 März 2006 15:00  
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chris22
Beiträge: 188 Registriert: November 2005 Ort: Berlin
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Vergiss den IGF-1 Spiegel  , schau dir mal Bilder von deinem tollen Haar im Alter von 6-10 Jahren an und dann heute, und dann schau dir die IGF-1 und IGFBP-3 Werte von damals und heute an, dann weisst du, wie gut hohes IGF-1 sein muss  ...
du brauchst deinen IGF-1 Spiegel nicht zu heben, ich weiss nicht mehr wie alt du genau bist, aber wenn du auch um die 20 Jahre alt bist, dann ist er schon weit über dem Wert, der für eine gesunde Matte erforderlich ist. Schau lieber darauf, dass sich deine Insulinsensivitaet nicht verringert und sie möglichst hoch ist, und dafür eignen sich KH gänzlich schlecht 
Gruss
Chris
[Aktualisiert am: Di., 28 März 2006 15:02]
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Re: kohlenhydrate/reiche /arme ernährung [Beitrag #16993 ist eine Antwort auf Beitrag #16867] :: Mi., 29 März 2006 12:40
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chris22
Beiträge: 188 Registriert: November 2005 Ort: Berlin
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pilos, in meinem Kopf irren andere Informationen umher
Effects of a fat-containing meal on sex hormones in men.
Meikle AW, Stringham JD, Woodward MG, McMurry MP.
Department of Internal Medicine, University of Utah School of Medicine, Salt Lake City.
The effect of a fat-containing meal on plasma sex steroid concentrations was investigated in normal men. After an overnight fast on two separate occasions, subjects ingested a liquid meal containing either a nonnutritive sweetener (control), or isocaloric meals of mixed calorie sources with either high-fat content or mixed carbohydrate and protein with minimal fat. The order of the meals was alternated. Blood samples were collected at 15-minute intervals and pooled each hour. Sampling began at 7:00 AM and the test meal was ingested at 8:00 AM. Sex steroids, including estrone, estradiol, testosterone, and dihydrotestosterone (DHT), sex hormone-binding globulin (SHBG) capacity, free testosterone concentration, and luteinizing hormone (LH) were determined by either specific radioimmunoassay or dialysis. The fat-containing meal, but not the nonnutritive or mixed carbohydrate and protein meal, resulted in a significant (P less than .01) reduction in total and free testosterone. Estrogens and luteinizing hormone were unaffected by either meal. This is the first documentation, to our knowledge, of the acute effect of a fat-containing meal on sex steroid concentrations in blood. Our observations suggest that a fat-containing meal reduces testosterone concentrations without affecting luteinizing hormone. This might indicate that fatty acids modulate testosterone production by the testes.
Proc Natl Acad Sci U S A. 1983 Dec;80(24):7646-9. Related Articles, Links
Nutrition-endocrine interactions: induction of reciprocal changes in the delta 4-5 alpha-reduction of testosterone and the cytochrome P-450-dependent oxidation of estradiol by dietary macronutrients in man.
Kappas A, Anderson KE, Conney AH, Pantuck EJ, Fishman J, Bradlow HL.
The in vivo biotransformations of drugs known to be metabolized by enzymes localized in the endoplasmic reticulum of liver can be greatly altered by diet in humans, as we have shown previously. Steroid hormones also are metabolized extensively by hepatic microsomal enzymes; therefore, we examined the possibility that testosterone and estradiol biotransformations, as assessed with radiolabeled tracer methods, could be influenced by dietary macronutrients. Normal males were fed a high-protein diet for 2 weeks, followed by a high-carbohydrate diet for an additional 2 weeks. The delta 4-5 alpha-reduction of testosterone was considerably diminished, while the cytochrome P-450-dependent hydroxylation of estradiol at the C2 position was substantially enhanced during ingestion of the high-protein diet as compared with the high-carbohydrate diet. These results indicate that dietary macronutrients can significantly alter major metabolic pathways for testosterone and estradiol in man. The mechanism by which reciprocal changes in the delta 4-5 alpha-reduction of testosterone and the cytochrome P-450-mediated oxidation of estradiol are produced by diets is not known. Similar changes in steroid delta 4-5 alpha-reduction and cytochrome P-450-dependent chemical oxidations have been observed in circumstances in which the mixed-function oxidase system in liver is induced by agents such as phenobarbital, hexachlorobenzene, dioxin, and polyhalogenated biphenyls. Thus, the alterations in steroid hormone metabolism produced by dietary macronutrients in man mimic those that can be produced by drugs and environmental chemicals.
Gruss
Chris
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