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Re: Häufiger Saunabesuch schädlich? [Beitrag #48567 ist eine Antwort auf Beitrag #48538] :: Di., 07 November 2006 23:13  
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tristan
Beiträge: 709 Registriert: November 2005
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| Milka schrieb am Die, 07 November 2006 21:26 |
ganz im gegenteil, saunabesuche sind gut für die haut ....
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sind sie nicht 
hitze ist schlecht für die haut. ob vom sonnenlicht, sauna oder sonst wo her. fakt. temperaturextrema generell sind schlecht, kaltes wasser, heißes wasser etc..
es entstehen mehr ROS, neutrophile usw., das komplette vasculäre system wird geschädigt, mit der zeit werden immunzellen hyperaktiv..
literatur gibt es viel dazu..
als beispiel:
J.Y.Seo,J.H.Chung,
Thermal aging: A newconcept of skin aging,(2006),
doi:10.1016/j.descs.2006.08.002
Department of Dermatology, Seoul National University College of Medicine, Seoul, South Korea
bLaboratory of Cutaneous Aging Research, Clinical Research Institute, Seoul National University Hospital, Seoul, South Korea
cInstitute of Dermatological Science, Seoul National University, Seoul, South Korea
Available 17 October 2006.
...
"Heat is a form of energy that may be transmitted in different ways, by direct contact-conduction, by circulating currents-convection, or by infrared radiation from a heated body [6]. Despite these different means of transmission, the final product (heat energy) is the same, manifesting as an increased skin temperature. Human skin is exposed daily to UV and infrared radiation. We found that the temperature of human skin, measured inside the dermis by a needle-type thermometer, was increased to 40–43 °C in direct summer midday sunlight within 15–20 min (personal observation). Radiant heat, as a by-product of stoves, furnaces, and similar devices, can also cause cutaneous changes similar to those found in chronically sun-exposed skin [7]. On more prolonged heat exposure, severe elastic fiber hyperplasia develops, which extends deeply into the dermis, in combination with the degeneration of dermal collagen [8] and [9]. Infrared radiation is converted to heat in the skin, and therefore, may have a more significant biological effect than previously realized."
...
"Recently, it was suggested that excessive matrix degradation by UV-induced matrix metalloproteinases (MMPs) secreted by various cells, including, keratinocytes, fibroblasts, and inflammatory cells, contributes substantially to the connective tissue damage that occurs during photoaging [13], [14] and [15]. UVB is known to induce the expressions of MMP-1, -3 and -9 in the normal human epidermis in vivo [13], and UVA is known to induce the expression of MMP-1 by dermal fibroblasts in vivo, and the expressions of MMP-1, -2, and -3 in culture [16].
The expression of MMPs proteins in the dermis of sun-exposed and sun-protected skin of elderly subjects is known to differ [14]. Photodamaged skin of the elderly shows higher MMP-1 protein expression, than the intrinsically aged skin in the same individual [14]. Sun-damaged forearm skin also shows significantly higher levels of active MMP-2, proMMP-2 and MMP-9 than intrinsically aged skin in the same individual. Thus, this resulting higher expression of MMPs in photoaged skin may cause severe collagen deficiency and wrinkling."...
"Heat shock is known to produce highly specific stress responses including the induction of a variety of heat shock proteins, which play a protective role against the harmful effects of stressful stimuli [20] and [21]. In contrast to its beneficial effects, several studies have shown that heat shock increases the expression of collagenase and stromelysin mRNA in synovial and dermal fibroblasts, and have suggested that heat shock-induced collagenase expression may contribute to connective tissue degradation in disease states"..
"Recently, we investigated the effects of heat on the expressions of tropoelastin in human skin in vivo and demonstrated, for the first time, that heat like UV, can increase tropoelastin mRNA and protein expression in the epidermis and in the dermis of human skin [42]. We also found that heat increased fibrillin-1 mRNA and protein expression in the epidermis, but decreased them in the dermis [42]. It has been also reported that heat-induced ROS may play a critical role in heat-induced tropoelastin expression [42]. Heat increased tropoelastin-positive fiber numbers, but reduces fibrillin-1-positive fibers in human skin, suggesting that the reduced fibrillin-1 expression in the dermis by heat might cause a reduction in the number of microfibril bundles in the upper dermis, and thus reduce tropoelastin deposition on microfibrils, leasing to abnormal elastic fiber assembly [42]. These results suggest that heat exposure regulates the synthesis of elastin and fibrillin, and affects the formation of elastic fibers in human skin. Therefore, like UV, heat is an important external stimulus, which modulates the formation of elastic fibers networks in human skin.
Heat treatment also increased MMP-12 mRNA and protein expression in human dermis in vivo [42]. It has been reported that heat increases the expression of collagenase and stromelysin mRNAs in synovial and dermal fibroblasts [22] and [23]. These heat-induced MMPs, including MMP-12, are capable of destroying the preexisting elastic fiber network and of degrading newly synthesized tropoelastin and fibrillin proteins, and thus contribute to the loss of elastic fibers and/or the accumulation of elastotic materials in photoaged skin."
"Heat treatment also increased the expression of IL-12 and IL-6 mRNA significantly in cultured dermal fibroblasts (Fig. 6). This heat-induced cytokines are involved in the regulation of extracellular matrix proteins such as collagen. For example, IL-6 increased the MMP-1 and –3 production and anti-IL-6 antibody inhibited the heat-induced MMP-1 and -3 expression in a dose-dependent manner as mentioned previously [24]. In summary, heat induced many cytokines and then, these cytokines regulate the metabolisms of extracellular matrix proteins metabolism in human skin."
"Conclusion. ...(1) heat exposure to human skin stimulates the expression of MMP-1, MMP-3, and MMP-12. This increased expression of MMPs by heat will degrade the extracellular matrix proteins such as collagen and elastic fibers in human skin; (2) heat also regulates the synthesis of elastin and fibrillin, affects the formation of elastic fibers in human skin, and contributes to the development of solar elastosis; (3) heat regulates the production of many cytokines, including TGF-β, IL-6, and IL-12 and then, these cytokines regulate the expressions of extracellular matrix proteins in human skin; (4) heat is an important physical stimulus on human skin, just like UV, which can cause angiogenesis in human skin. Therefore, in addition to sunscreen, new strategies to block heat-induced skin aging (thermal aging) need to be developed to more effectively prevent skin aging "
was ist daran gut  , das ist katasprophe, ai..
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Re: Häufiger Saunabesuch schädlich? [Beitrag #48879 ist eine Antwort auf Beitrag #48764] :: Do., 09 November 2006 19:33
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| tristan schrieb am Don, 09 November 2006 02:10 |
| tvtotalfan schrieb am Mit, 08 November 2006 20:42 |
Wie ist es wenn man täglich sehr heiss duscht ?
Meine Freundin tut das immer, das Wasser ist dabei so heiss dass ich nie zusammen mit ihr drunter stehe 
Auch schlecht für die Haut ?
Die ist bei der aber suuuper 
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wie oben geschrieben, jegliche Hitze schadet der Hautstruktur und verschnellert die Alterung der Haut. das ist ein andauernder Prozess, das sieht man nicht sofort...
ich denk mir das nicht aus, das könnt ihr doch auch nachlesen...
soll schon was heißen wenn du nicht mit duschen möchtest, das muss ja gefährlich sein. eine verbrennung reicht was? sorry
lg
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noin.....sag das nicht mit der hitze.....ich bin ein heißduscher.....och mist! 
[Aktualisiert am: Do., 09 November 2006 19:33]
oral: 1,25mg Finasterid, 2g MSM, eine Vitamintablette, sporadisch Zink.
topisch: morgens: 5% Minoxidil von Genhair
abends: 15% Minoxidil von Genhair
gemischt mit 2% Flutamid
Zwischendinn das Proctor Zeug
Fazit: Ganz gute Erfolge
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Den Beitrag einem Moderator melden
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